- During hypertensive left ventricular hypertrophy, what does the ventricular wall stiffness impair and what does this cause
- What are the poor prognostic indicators for Tachyarrhythmia?
- Depends on the cause of anemia. Involves replacement of missing nutrients needed for the production of hemoglobin and proper red blood cell development
- Blocks Angiotensin II from attaching to receptor sites and causing vasoconstriction. Reduces blood pressure
- Interrupt Vitamin K production of cofactors that help blood to clot. Decrease Vit. K in the body. Block cofactors thrombin and fibrin that are required to form clots
- Dilates arteries and decreases heart rate. Results in decreased blood pressure
- IFU: Congestive Heart Failure (CHF) => increase blood volume causing workload on the heart
- - Microzide (hydrochlorothiazide)- Aldactone (spironolactone)- Lasix (furosemide)
- - Nitrostat (nitroglycerin)- Nitrolingual (nitroglycerin)
- Relaxes smooth muscle around blood vessels. Dilates arteries/veins. Decreases O2 used by cardiac muscle
- - Amicar (aminocaproic acid)- Cyklokapron (tranexamic acid)
- - Apresoline (hydralazine)- IMDUR (isosorbide mononitrate)
- - Diovan (valsartan)- Benicar (olmesartan)
- Deactivates the NA+/K+ ATPase pump and activates the Na+/Ca+ exchanger. Results in increased Ca+ inside of the cell = improved heart contractions, circulation, rhythm and promotes removal of excess fluid by kidneys
- Increases urinary excretion of Na+, H2O, and sometimes K+. Less fluid in the body helps reduce blood pressure. +There are 3 subclasses*
- Relaxes smooth muscles of blood vessels. Dilates arteries/veins = decreased resistance on the heart making it easier to pump
- Block potassium channels on cardiac muscle cells and keeps potassium from leaving the cells. Increased potassium inside of cells = normal heart rhythm
- Works on kidneys to prevent conversion of Angiotensin I to Angiotensin II. Decreases Na+ and H2O retention => decreased blood pressure
- Inhibits the secretion of triglycerides from the liver. Reduces blood triglyceride levels and activates fat enzymes (lipoprotein lipase) to aid with the breakdown of fat
- Slows down the liver's production of cholesterol by blocking HMG-CoA reductase enzyme in the liver