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Black widow spider venom is thought to disrupt the functioning of nerve terminals by a. proteolytic cleavage of SNARE proteins. b. circumventing the calcium-regulatory step of exocytosis to promote massive exocytosis. c. binding to all molecules of synapsin, synaptotagmin, and synaptophysin and thereby preventing their normal functioning. d. punching holes in vesicles and thereby causing release of their contents into the cytosol. e. blocking calcium channels.
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